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Transcriptional control of meiotic recombination by the DREAM complex

Created on 04 Nov 2025

Authors

Tuncay, H., Ma, X., Lang, L., Boewer, F., Latrasse, D., Benhamed, M., Schnittger, A.

Abstract

Meiotic chromosome segregation and recombination, essential for sexual reproduction and evolution, require a specialized gene set. However, how meiotic gene expression is controlled remains largely unclear in multicellular eukaryotes. Here, we identify the DREAM complex as a transcriptional regulator of meiotic recombination in Arabidopsis. We show that the DREAM complex localizes to meiotic chromosomes, and that reduced DREAM function disrupts recombination and causes sterility. Meiocyte-specific transcriptomics combined with genome-wide binding analyses revealed that recombination genes, including the crossover-promoting helicase MER3, are direct DREAM targets. Notably, bypassing DREAM regulation of MER3 partially restores recombination in DREAM-deficient plants. Our findings establish the DREAM complex as a major meiotic regulator. Given that reduced DREAM function causes reproductive defects in species such as Drosophila and mice, this role may be conserved across multicellular eukaryotes.

Preprint server: bioRxiv
The authors list and abstract were imported from bioRxiv on 04 Nov 2025.

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