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Repurposed anticancer drugs disrupt cancer-like traits of Theileria annulata and suppress Leishmania donovani.

Created on 14 Jul 2026

Authors

Samoel Lareb, Madhusmita Subudhi, Akash Suresh, Vasundhra Bhandari, Umarani Brahma, Paresh Sharma

Published in

Cell communication and signaling : CCS. Jul 13, 2026. Epub Jul 13, 2026.

Abstract

Protozoan parasites remain a major threat to global health, affecting both humans and livestock. Theileria annulata, an apicomplexan parasite of cattle, transforms host leukocytes into a cancer-like state, while Leishmania donovani causes visceral leishmaniasis in humans. Given their reliance on host signaling and metabolic pathways, we hypothesized that FDA-approved anticancer drugs could be repurposed as antiparasitic agents. A library of 436 compounds was screened against T. annulata, yielding 130 hits at 10 µM, of which 63 exhibited minimal cytotoxicity toward healthy PBMCs. Secondary screening at 1 µM identified 28 potent inhibitors with > 50% activity. These compounds span diverse scaffolds, including taxanes, nucleoside analogues, antifolates, proteasome inhibitors, and kinase modulators, and target critical pathways such as apoptosis, autophagy, DNA damage response, cytoskeletal dynamics, NF-κB signalling, and tyrosine kinase activity. Notably, floxuridine, masitinib, paclitaxel, cabazitaxel, vinorelbine, maytansine, and bortezomib also suppressed L. donovani, highlighting cross-species efficacy. Target mapping revealed that the majority of active molecules disrupt hallmarks of cancer and parasite-transformed cells, including metabolic reprogramming, genome instability, and invasive phenotypes. Collectively, these findings underscore the value of oncology-derived drug libraries for antiparasitic discovery and identify promising candidates for further evaluation against T. annulata and L. donovani.

PMID:
42443891
Bibliographic data and abstract were imported from PubMed on 14 Jul 2026.

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