Hiring in life sciences? Share your open positions with our professional community. Read more Close

Advertisement

miR172-AP2 Controls Age-Dependent Regeneration Competence in Populus.

Created on 04 Aug 2026

Authors

Kaiwen Lyu, Abdul Azeez, Victor Busov

Published in

Plant biotechnology journal. Aug 03, 2026. Epub Aug 03, 2026.

Abstract

Plants maintain remarkable regenerative potential, yet this capacity declines with developmental age, limiting clonal propagation and biotechnological applications. Here, we show that the miR172-AP2 module serves as a central determinant of regeneration competence in Populus. Regenerative capacity declines progressively along the shoot developmental gradient, correlating with elevated miR172 and reduced AP2 transcript levels. Functional analyses demonstrate that AP2 promotes both shoot and root regeneration by directly activating downstream transcriptional programmes, including AINTEGUMENTA-LIKE1, BIG LEAF and LIKE APETALA1, which regulate organogenic growth and developmental competence. Elevated miR172 or cytokinin signalling represses AP2, reducing regenerative outcomes, indicating that hormonal and developmental signals converge on this module. Genome-wide analyses reveal that AP2 coordinates a broad network of genes controlling wound response, hormone signalling and cell fate specification. Notably, enhanced regeneration via AP2 occurs without detectable developmental abnormalities, highlighting the practical potential of this module. Collectively, these findings uncover a mechanistic link between developmental phase identity and regeneration, revealing a conserved regulatory module that may be leveraged to enhance regeneration across plant species, with broad implications for propagation, tissue culture and biotechnology.

PMID:
42545830
Bibliographic data and abstract were imported from PubMed on 04 Aug 2026.

Read full publication at:
Please sign in to see all details.

Advertisement

Stats

  • Community rating n/a 0 votes
  • Reviewers' rating n/a 0 votes
  • Your rating

1-terrible, 9-excellent. How would you rate this publication? Sign in in to submit your rating.

  • Recommendations n/a n/a positive of 0 vote(s)
  • Views 4
  • Comments 0

Recommended by

  • No recommendations yet.

Post a comment

You need to be signed in to post comments. You can sign in here.

Comments

There are no comments yet.

Advertisement