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DEPACE-seq enables high-fidelity, single-nucleotide-resolution mapping of abasic sites and characterization of dynamic and long-lived AP landscapes in mammalian genomes.

Created on 15 Sep 2026

Authors

Hangrui Wu, Jiayu Wang, Chenxu Zhu, Xinyi Li, Tengyuan Zhang, Qifei Zeng, Meiping Zhao

Published in

Nucleic acids research. Volume 54. Issue 14. Jul 17, 2026.

Abstract

High-fidelity single-nucleotide-resolution mapping of abasic (AP) sites in mammalian genomes remains technically challenging due to low abundance and high background. Here we present DEPACE-seq (Dual-End PAB-Conjugated Endo IV-Cleaved Sequencing), a robust and easy-to-implement method for precise genome-wide profiling of AP sites. DEPACE-seq employs an N-pyrrolyl-alanine-2,2'-(ethylenedioxy)bis(ethylamine)-biotin (PAB) probe that selectively conjugates AP sites via a mild Pictet-Spengler reaction. The resulting PAB-AP adducts are efficiently and specifically cleaved by endonuclease IV, while remaining inert to other aldehyde-containing DNA bases. Independent library construction from both cleavage ends and the intersection of the resulting signals enable high-confidence identification of AP sites at single-nucleotide resolution. Application of DEPACE-seq across different cell types and damage conditions enabled high-confidence characterization of two AP-site populations: long-lived sites enriched in satellite and intergenic regions, and repair-intermediate sites induced by transient damage and enriched in transcriptionally active regions, both exhibiting cell-type-dependent features. Molecular dynamics simulations further elucidate how endonuclease IV accommodates bulky PAB adducts. Together, DEPACE-seq provides a robust platform for high-confidence, single-nucleotide-resolution mapping of AP sites in mammalian genomes, enabling systematic investigations of genome instability, DNA repair, aging, and cancer.

PMID:
42559686
Bibliographic data and abstract were imported from PubMed on 15 Sep 2026.

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